Basic valvular lesions on 2D and color
Stem
Monitors and echo station. A 76-year-old man, 80 kg, with a loud systolic murmur that was never worked up, becomes hypotensive after induction for a hip fracture repair, and a TEE probe is placed. The first loop is a midesophageal aortic valve short-axis view: three heavily calcified cusps that barely separate, with a planimetered opening of 0.8 cm². The second is a midesophageal long-axis view with color: a thick, concentrically hypertrophied LV and a mosaic jet above the valve in systole.
Question
What is the lesion, how severe is it, and what are your hemodynamic goals?
Ideal answer
HeadlineSevere aortic stenosis: calcified, restricted cusps, planimetered area below 1.0 cm².1 My goals: sinus rhythm, rate 60–80, full preload, normal-to-high pressure with phenylephrine early.
- View, then leaflets on 2D: midesophageal aortic valve short-axis; cusps thickened, calcified and restricted.
- The chamber that pays: the long-axis shows concentric hypertrophy, paying the pressure load.
- Color, Nyquist 50–70 cm/s, gain just below speckle:2 turbulent mosaic flow above the valve in systole, no diastolic jet into the outflow tract.
- Grade on leaflet opening, not color turbulence, because turbulence does not measure severity.
- Regurgitant instead: the opposite, a normal-to-fast rate and lower afterload, to cut the regurgitant volume.
Breaking down the answer
The outline limits valve disease to what I can see on 2D and color flow; there is no pulsed or continuous-wave Doppler on the exam.3 It tests recognition from structure and jet, a rough grade, and the goals.
| Lesion | Best view | 2D and color findings | Severe when |
|---|---|---|---|
| Aortic stenosis | ME AV short-axis, ME long-axis | Calcified cusps, restricted opening, concentric LVH, turbulent flow above the valve | Planimetered area <1.0 cm²1 |
| Aortic regurgitation | ME long-axis | Diastolic jet into the LVOT from the cusps, dilated LV in chronic disease | Central jet width ≥65% of LVOT; vena contracta >6 mm2 |
| Mitral regurgitation | ME four-chamber, two-chamber, long-axis | Systolic jet into the LA; prolapse or flail leaflet; eccentric jets hug the wall | Vena contracta ≥7 mm; central jet >50% of LA area, or an eccentric wall-impinging jet2 |
| Mitral stenosis | ME four-chamber, long-axis | Thickened leaflets, "hockey-stick" anterior leaflet doming, big LA, spontaneous contrast | Severe restriction with marked LA enlargement |
| Tricuspid regurgitation | ME four-chamber, RV inflow-outflow | Systolic jet into the RA, dilated RV and annulus | Vena contracta >7 mm; central jet >50% of RA; flail or no coaptation2 |
- Vena contracta over jet area — jet area depends on gain, the Nyquist limit and loading; the narrowest neck is far less load-dependent, so it is my main severity grade.
- Eccentric jets are underestimated — a jet hugging the atrial wall loses momentum and looks smaller than a central jet of the same severity. The prolapsing or flail leaflet is usually opposite the jet direction, which tells the surgeon which leaflet to repair.2
- Why the AS goals — the atrial kick and diastolic time fill a stiff ventricle, and a fall in diastolic pressure starves the hypertrophied subendocardium; goals stated after the lesion show judgment.
- Regurgitant lesions — rate 80–100 and lower afterload shorten the time and lower the gradient driving backward flow; I avoid bradycardia and pure vasoconstrictors.
- Mitral stenosis goals — a slow rate to lengthen diastolic filling, rate control of atrial fibrillation, and no hypoxia, hypercarbia or acidosis, which raise pulmonary pressure on a hard-working RV.
Follow-up question
Next scenario: after a mitral valve repair, the patient is hypotensive. The loop shows a mosaic color jet in the LV outflow tract in systole, a posteriorly directed mitral regurgitant jet, and the anterior leaflet swinging toward the septum. What is this, and how do you treat it?
Follow-up answer
HeadlineSystolic anterior motion with dynamic LV outflow obstruction; I tell the surgeon the finding and mechanism, because the instinctive treatment worsens it.
Stop inotropes, give volume, raise afterload with phenylephrine, slow the rate with esmolol, lighten the dilators. I reassess outflow jet and regurgitation on the next loop; if obstruction persists, the surgeon revises the repair. The same picture appears in an underfilled patient with a septal bulge. The other variant, acute severe MR from papillary muscle rupture after an infarct: a flail leaflet, wide jet, hyperdynamic LV; I lower afterload, support with an inotrope, and call for an intra-aortic balloon pump and surgery.
Pitfalls that cost points
- Grading regurgitation on jet area with the Nyquist limit set too low.
- Calling a stenosis severe from color turbulence alone, ignoring leaflet opening.
- Offering a Doppler gradient the exam does not ask for and cannot show.
- Treating hypotension from systolic anterior motion with epinephrine.
Rapid-fire
Q: Why does color mosaic appear? A: Velocities above the Nyquist limit alias, so high-speed turbulent flow shows as a mix of colors.
Q: Why are eccentric jets underestimated? A: The Coanda effect pulls the jet along the atrial wall, so its visible area shrinks while the regurgitant volume does not.
Q: Why does an empty, fast heart worsen systolic anterior motion? A: A small LV cavity and high outflow velocity drag the anterior leaflet into the outflow tract by flow drag and Venturi forces.
Q: Which view shows the aortic valve as three cusps? A: The ME aortic valve short-axis at about 25–45°.4
Numbers to know
| Item | Value |
|---|---|
| Color Nyquist limit | 50–70 cm/s2 |
| Severe AS area | <1.0 cm²1 |
| Severe AR vena contracta | >6 mm2 |
| Severe MR vena contracta | ≥7 mm2 |
| AS heart-rate goal | 60–80 bpm |
| Regurgitant heart-rate goal | 80–100 bpm |
Links
- The 11 TEE views, Aortic stenosis, Mitral regurgitation and mitral stenosis, Hypertrophic obstructive cardiomyopathy
- Atlas: The 11 TEE views
- 2020 ACC/AHA Guideline for the Management of Valvular Heart Disease; ASE Recommendations for Noninvasive Evaluation of Native Valvular Regurgitation (2017)
Sources
- 1.Echocardiographic Assessment of Valve Stenosis: EAE/ASE Recommendations for Clinical Practice · EAE/ASE, 2009 · asecho.orgASE note: use ACC/AHA staging for mitral stenosis instead of the 2009 values.
- 2.Recommendations for Noninvasive Evaluation of Native Valvular Regurgitation · ASE/SCMR, 2017 · asecho.orgNyquist 50-70 cm/s; severity tables for MR, AR, TR.
- 3.APPLIED Exam Objective Structured Clinical Examination (OSCE) Content Outline · ABA · theaba.org
- 4.Guidelines for Performing a Comprehensive Transesophageal Echocardiography Examination · ASE/SCA, 2013 · asecho.org
Facts on this page were checked against these sources on Oct 14, 2026. Guidelines change; confirm doses against current institutional protocols.